LETTER TO THE EDITOR |
Dementia and Neurocognitive Disorders 2017: 16: 2: 54-55 |
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Unusual FDG-PET Findings in Traumatic Brain Injury; Did Traumatic Brain Injury Provoke Rapid Progression of Alzheimer’s Disease? |
Inha Hwang, Sang-Won Ha, Jeong Ho Han |
Department of Neurology, Veterans Health Service Medical Center, Seoul, Korea |
Unusual FDG-PET Findings in Traumatic Brain Injury; Did Traumatic Brain Injury Provoke Rapid Progression of Alzheimer’s Disease? |
Inha Hwang, Sang-Won Ha, Jeong Ho Han |
Department of Neurology, Veterans Health Service Medical Center, Seoul, Korea |
Traumatic brain injury (TBI) is common, and is often the leading cause of disability and death. Complications after TBI include increased risk for chronic central nervous system disease, such as Alzheimer’s disease (AD). However, the pathophysiology relating acute injury to neurodegeneration is unclear. Here we present a case of a patient whose cognition declined after TBI, and whose 18F fluorodeoxyglucose positron emission tomography scan showed an AD pattern. |
Key Words: traumatic brain injury fluorodeoxyglucose positron emission tomography Alzheimers disease |
대한치매학회지 (Dementia and Neurocognitive Disorders) |